美国binaxnow试剂沼泽藏有军团菌病菌快速检测卡

沼泽藏有军团菌病菌快速检测卡

广州健仑生物科技有限公司

广州健仑长期供应：军团菌、诺如病毒、流感病毒等传染病系列的快速检测试剂盒。

军团菌的检测试剂盒包括：军团菌尿液抗原检测试剂盒、军团菌抗体快速检测卡（胶体金法）、军团菌抗原快速检测卡（胶体金法）、军团菌水样检测试剂盒、军团菌乳胶凝集试剂盒（军团菌诊断血清）、嗜肺军团菌核酸荧光PCR检测试剂盒。

我司还提供其它进口或国产试剂盒：包括传染病系列、免疫组化系列、诊断血清等产品。

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沼泽藏有军团菌病菌快速检测卡

实验步骤

1） 将所有的材料和样品都平衡至室温（2-30℃）

2） 将所有的检测卡从密封的试剂袋中取出。

3） 将样品点滴器垂直置于样品孔上方，向样品孔中加入3滴样品（120-150ul）。

4） 10分钟内读取结果，强阳性样品可能会早点出现结果。

注意：10分钟后读取的实验结果可能会不准确。

结果说明

阳性结果：检测线区域出现明显的粉色条带，另外质控线区域出现粉色条带。

阴性结果：检测线区域不显色，质控线区域出现明显的粉色条带。

无效结果：靠近检测线的质控线在加样品后15分钟内不可见的话，则实验结果无效。

7、产品特点
★操作简便，无需其它仪器和试剂，易于在各级医院推广；
★反应迅速，5分钟内即可得到结果；
★结果清晰，易于判定；
★敏感度高，特异性强。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司

【市 场 部】    杨永汉

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【腾讯Q Q】 2042552662

【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

该分段法各段的解剖分界明确，共 分为七段：C1颈段(Cervical segement)，C2岩段（Petrous  segment），C3破裂（孔）段（Lacerum segment），C4海绵窦段 （Cavenous segement），C5床段（Clinoidal segment），C6眼段 （Opt细菌almic segement）和C7交通段（Communicating segment ）[1]  。C1颈段：颈段起于颈总动脉分叉水平，终止于颈动脉管颅外口。C2岩 段：这段颈内动脉位于颈动脉管内，起于颈动脉管颅外口，终止于破 裂孔后缘。C3破裂（孔）段：破裂段起于颈动脉管末端，动脉越过孔 部，但不穿过这个孔，在破裂孔的垂直管内上升，向着海绵后窦，止 于岩舌韧带上缘。C4海绵窦段：此段始于岩舌韧带上缘，止于近侧硬 膜环。C5床段：此段起于近侧硬膜环，止于远侧硬膜环。C6眼段：该 段起于远侧硬膜环，止于后交通动脉起点的紧近侧。C7交通段：交通 段起于紧靠后交通动脉起点的近侧，止于颈内动脉分叉处。动眼神经 （oculomotor nerve）为运动性神经，含有躯体运动和内脏运动两种 纤维。躯体运动纤维起于中脑动眼神经核，一般内脏运动纤维起于动 眼神经副核。动眼神经自脚间窝出脑，紧贴小脑幕缘及后床突侧方前 行，进入海绵窦侧壁上部，再经眶上裂眶，立即分为上、下两支。上 支细小，支配上直肌和上睑提肌。下支粗大，支配下直、内直和下斜 肌。由下斜肌支分出一个小支叫睫状神经节短根，它由内脏运动纤维 （副交感）组成，进入睫状神经节交换神经元后，分布于睫状肌和瞳 孔括约肌，参与瞳孔对光反射和调节反射。动眼神经麻痹时，出现上眼睑下垂，眼球向内、向上及向下活动受限 而出现外斜视和复视，并有瞳孔散大，调节和聚合反射消失。常见的 病细菌有动眼、滑车与外展神经本身炎症而致的麻痹，急性感染性多 发性神经炎，继发于头面部急、慢性炎症而引起海绵窦血栓形成。

The anatomical boundaries of each segment of the segmentation method are clear and divided into seven sections: Cervical segement, Petrous segment, Lacerum segment, Cavenous segment, segement, Clinoidal segment, Optics almic segement and C7 Communicating segment [1]. C1 neck: the neck from the common carotid artery bifurcation level, terminating in the carotid artery cranial mouth. Section C2: This section of the internal carotid artery located in the carotid artery, the carotid artery in the cranial mouth, terminating in the posterior edge of the rupture. C3 Rupture (Pore): The rupture begins at the end of the carotid artery. The artery passes through the hole but does not pass through this hole. It rises in the vertical tube of the ruptured hole toward the posterior sinus of the sponge, stopping at the upper edge of the tongue. C4 cavernous sinus segment: This segment began in the upper edge of the tongue ligament, ending in the proximal dural ring. C5 bed segment: This section from the proximal dural ring, ending in the distal dural ring. C6 segment: the segment from the distal dural ring, just after the start of the posterior communicating artery close to the side. C7 traffic section: the traffic section close to the starting point of the artery immediay after the carotid artery at the bifurcation. Oculomotor nerve is a motor nerve that contains both somatic and visceral motility fibers. Somatic motor fibers start in the midbrain optic nerve nucleus, the general movement of visceral fibers from the oculomotor nerve nucleus. Occurred from the foot of the oculomotor nerve, close to the cerebellar margin and the posterior lateral approach, into the upper cavernous sinus wall, and then through the supraorbital fissure, immediay divided into upper and lower two. Upper small, dominate the rectus and levator muscle. Under the branch thick, dominated the next straight, straight and lower oblique. From the inferior oblique muscular branch of a small branch called the ciliary ganglion short root, which consists of visceral motility fibers (parasympathetic), into the ciliary ganglion neurons, distributed in the ciliary muscle and pupil sphincter, involved in pupillary pairs Light reflection and reflection adjustment. Oculomotor nerve paralysis, the upper eyelid ptosis, eyeball inward, upward and downward mobility constraints appear exotropia and diplopia, and dilated pupils, regulation and polymerization disappear. Common disease bacteria have moving eyes, block and outreach nerve inflammation itself caused by paralysis, acute infectious polyneuritis secondary to head and face acute and chronic inflammation caused by cavernous sinus thrombosis, supraorbital fissure and orbital apex Syndrome, intracranial aneurysms, intracranial tumors, other such as tuberculosis, mold, syphilis and purulent inflammation caused by skull base meningitis. Due to different bacteria, the pathogenesis is also different, such as direct compression of the tumor caused by the primary inflammation.