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【公司名称】 广州健仑生物科技有限公司
【】    杨永汉 
【】 
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乍看之下，Davies似乎不像“癌症战争”中的战士，他是物理学出身，而非生物医学。但是，大约7年前，他被邀请在亚利桑那州建立一个新机构——由国立癌症研究所资助的12所机构中的1个，以便将物理学家和肿瘤学家联合在一起，发现该疾病的新视角。“我们被要求从上到下重新思考癌症。”Davies说。
随后，Davies与澳大利亚国立大学天体生物学家Charley Lineweaver和英国伦敦健康科学中心肿瘤学家Mark Vincent展开合作，提出了“返祖现象”模式，将癌症定位为古老“预编”特性的重新表达。在上个月发表于《生物学论文集》的新研究中，该研究小组指出，因为癌症出现在许多动物、植物和人类中，那么它必须从亿万年前开始进化，那时生物拥有共同的单细胞祖先。
在那时，细胞受益于永生，或无限增殖能力，正如癌症那样。但当复杂的多细胞生物开始出现，“‘永生’被转包给卵子和抗原抗体。”Davies说，不涉及繁衍的体细胞不再需要这种机能。
该研究小组提出的假设是，当健康细胞面临环境威胁时，例如辐射或生活因素，细胞能够回复到“预编的安全模式”。这样一来，细胞会抛弃更高的机能，并将它们的休眠能力切换至增殖能力，以便存活下来。“癌症是一种自动防障功能，”Davies提到，“一旦这个子程序被触发，就会冷酷地运行该程序。”
9月11日，在英国帝国理工学院举办的一个医学工程会议上，Davies描述了一系列基于这种返祖现象的癌症疗法。Davies指出，与简单地攻击癌症复制能力不同，该模型揭示了“癌症的阿喀琉斯之踵”。例如，如果该理论正确，那么癌症进化初期地球的环境更酸且氧气更稀薄。因此，研究人员预测，利用高水平氧气和在饮食中加入还原糖以降低酸性，能够抑制肿瘤并引起肿瘤收缩。

At first glance, Davies does not seem like a warrior in the "cancer war", he is a physics background, rather than biomedical. However, about seven years ago he was invited to establish a new agency in Arizona, one of 12 institutions funded by the National Cancer Institute, to bring together physicists and oncologists to discover the disease New perspective. "We were asked to rethink the cancer from top to bottom," Davies said.
Davies then collaborated with astrobiologist Charley Lineweaver of the Australian National University and Mark Vincent, an oncologist at the London Health Science Center in the UK, to propose a "return to ancestor phenomenon" model that positions cancer as a re-expression of the ancient "pre-programmed" nature. In a new study published in the Proceedings of Biology last month, the team pointed out that because cancer appears in many animals, plants and humans, it must evolve hundreds of millions of years ago when the creatures have common Single cell ancestor.
At that time, cells benefit from eternal life, or infinite proliferative capacity, just as cancer does. But when complex multicellular organisms begin to emerge, "Immortality" is subcontracted to the egg and antigenic antibodies, Davies said, not involving multiplying somatic cells that are no longer needed for this function.
The team's hypothesis is that when healthy cells are exposed to environmental threats, such as radiation or life, cells can revert to a "pre-programmed security model." As a result, cells discard higher functions and switch their dormancy to proliferative capacity in order to survive. "Cancer is an automatic barrier," Davies said. "Once the subroutine is triggered, it runs coldly."
On September 11, at a medical engineering conference organized by Imperial College London, Davies described a series of cancer therapies based on this anteversion phenomenon. Davies points out that unlike a simple attack on cancer replication, the model reveals "the Achilles heel of cancer." For example, if the theory is correct, the Earth's environment is more acidic and oxygenier at the beginning of cancer's evolution. Therefore, the researchers predicted that the use of high levels of oxygen and added to the diet reducing sugar to reduce acidity, can inhibit the tumor and cause tumor shrinkage.